INFRA-E-MOTION activity monitoring systems [Site Map] [Site Map]
[Deutsche Version] [Deutsche Version]
[Home]
[News]
[Mouse-E-Motion]
[Products & Prices]
[Download]
[Support]
[» References]
[Contact Us]
References

The MOUSE-E-MOTION data logger is currently in use at the following institutions:

Germany:
  • Animal Facility of the University Hospital Eppendorf, University of Hamburg

  • Institute for Neural Signal Transduction, Center for Molecular Neurobiology, University of Hamburg

  • Institute for Molecular Neuropathobiology, Center for Molecular Neurobiology, University of Hamburg

  • Institute for Biosynthesis of Neural Structures, Center for Molecular Neurobiology, University of Hamburg

  • Department for Behavioural Biology, Institute for Neuro- and Behavioural Biology, University of Muenster

  • Department of Psychopharmacology, Central Institute of Mental Health, University of Mannheim

  • Department of Anatomy II, University Hospital of the Johann Wolfgang Goethe University, Frankfurt/M.

  • Department of Molecular Neurobiology, Institute of Biology, Free University of Berlin

  • Molecular Neurobiology workgroup, Department of Normal and Pathological Physiology, Philipps University Marburg


International:
  • Department of Neurobiology, University of Massachusetts Medical School, Worcester, Massachusetts, USA

  • Department of Animal Ecology, Institute of Zoology and Hydrobiology, University of Tartu, Estonia

  • Alcohol Research Centre, National Public Health Institute, Helsinki, Finland

Literature

The MOUSE-E-MOTION data logger is mentioned in the following scientific publications:

[1]   Freitag S, Schachner M, Morellini F.
Behavioral alterations in mice deficient for the extracellular matrix glycoprotein tenascin-R.
Behavioural Brain Research; 2003;145:189-207.
[2]   Schmidt A, Marescau B, Boehm EA, Renema WK, Peco R, Das A, Steinfeld R, Chan S, Wallis J, Davidoff M, Ullrich K, Waldschutz R, Heerschap A, De Deyn PP, Neubauer S, Isbrandt D.
Severely altered guanidino compound levels, disturbed body weight homeostasis, and impaired fertility in a mouse model of guanidinoacetate N-methyltransferase (GAMT) deficiency.
Human Molecular Genetics; 2004;13:905-921.
[3] Peters HC, Hu H, Pongs O, Storm JF, Isbrandt D.
Conditional transgenic suppression of M channels in mouse brain reveals functions in neuronal excitability, resonance and behavior.
Nature Neuroscience; 2005; Jan 8(1):51-60.
[4] Deacon RMJ, Brook RC, Meyer D, Haeckel O, Ashcroft FM, Miki T, Seino S, Liss B.
Behavioral phenotyping of mice lacking the KATP channel subunit Kir6.2.
Physiology & Behavior; 2006; 87:723-733.
[5] Löhmus M, Sundström LF, Moore RF.
Non-invasive corticosterone treatment changes foraging intensity in red-eyed vireos Vireo olivaceus.
Journal of Avian Biology; 2006; 37:523-526.
[6] Morellini F, Schachner M.
Enhanced novelty-induced activity, reduced anxiety, delayed resynchronization to daylight reversal and weaker muscle strength in tenascin-C-deficient mice.
European Journal of Neuroscience; 2006; 23:1255-68.
[7] Vengeliene V, Leonardi-Essmann F, Perreau-Lenz S, Gebicke-Haerter P, Drescher K, Gross G, Spanagel R.
The dopamine D3 receptor plays an essential role in alcohol-seeking and relapse.
The FASEB Journal; 2006; 20:2223-2233.
[8] Füllgrabe MW, Vengeliene V, Spanagel R.
Influence of age at drinking onset on the alcohol deprivation effect and stress-induced drinking in female rats.
Pharmacology, Biochemistry and Behavior; 86 (2007); 320-326.
[9] Vengeliene V, Heidbreder CA, Spanagel R.
The effects of lamotrigine on alcohol seeking and relapse.
Neuropharmacology; 2007 Dec; 53(8):951-7.
[10] Sawallisch C, Berhörster K, Disanza A, Mantoani S, Kintscher M, Stoenica L, Dityatev A, Sieber S, Kindler S, Morellini F, Schweizer M, Boeckers TM, Korte M, Scita G, Kreienkamp HJ.
The insulin receptor substrate of 53 kDa (IRSp53) limits hippocampal synaptic plasticity.
The Journal of Biological Chemistry; 2009 Apr 3; 284(14):9225-36.
[11] Von der Goltz C, Vengeliene V, Bilbao A, Perreau-Lenz S, Pawlak CR, Kiefer F, Spanagel R.
Cue-induced alcohol-seeking behaviour is reduced by disrupting the reconsolidation of alcohol-related memories.
Psychopharmacology (Berl). 2009 May 6.
[12] Pfeffer M, Müller CM, Mordel J, Meissl H, Ansari N, Deller T, Korf HW, von Gall C.
The mammalian molecular clockwork controls rhythmic expression of its own input pathway components.
The Journal of Neuroscience; 2009 May 13; 29(19):6114-23.

[Top of the page] [Top]
Last updated on Jun 14, 2009.
[Top] [Top of page]